Associate Professor | School of Health and Sciences
Ext. 5220
franciscom.arencibia@sagrado.edu
Dr. Arencibia-Albite completed a Bachelor's degree in Exercise Science and Health Promotion at the Universidad del Sagrado Corazón Heart. Later, at that institution, he completed degrees in Mathematics and Biology. In addition, he holds a master's degree in Physiology from the School of Medicine of the Central University of the Caribbean in Bayamón. In 2013, he completed doctoral studies in Physiology and Biophysics at the Medical Sciences Campus of the University of Puerto Rico (RCM). He has worked as a researcher in the areas of biophysics, electrophysiology and computational neuroscience at the Medical Sciences Campus of the University of Puerto Rico (RCM). He has been recognized in Sagrado with the award for excellence in teaching on two occasions.
Research Projects
Department of Physiology and Biophysics
University of Puerto Rico, Medical Sciences Campus, San Juan, PR
Laboratory of Dr. Carlos Jiménez (2015-2017) Postdoctoral Fellow
Department of Physiology and Biophysics
University of Puerto Rico, Medical Sciences Campus, San Juan, PR
Dr. Carlos Jimenez's Lab (2013-2015) Research Assistant
Department of Physiology and Biophysics
University of Puerto Rico, Medical Sciences Campus, San Juan, PR
Dr. Carlos Jimenez's Laboratory (2008-2013) Graduate Student
Department of Physiology
Central University of the Caribbean, Bayamón, PR
Dr. Carlos Jimenez's Laboratory (2000-2004) Graduate Student
Peer-reviewed publications
Arencibia-Albite F. (2022). The energy balance theory is an inconsistent paradigm. Journal of theoretical biology, 550, 111240. https://doi.org/10.1016/j.jtbi.2022.111240
Arencibia-Albite, F., & Jiménez-Rivera, CA (2021). Computational and theoretical insights into the homeostatic response to the decreased cell size of midbrain dopamine neurons. Physiological reports, 9(2), e14709. https://doi.org/10.14814/phy2.14709
Arencibia-Albite F. (2020). Serious analytical inconsistencies challenge the validity of the energy balance theory. Heliyon, 6(7), e04204. https://doi.org/10.1016/j.heliyon.2020.e04204
Arencibia-Albite F, Vázquez R, Jiménez-Rivera CA. (2017) Cocaine sensitization increases subthreshold activity in dopamine neurons from the ventral tegmental area. J Neurophysiol. 117(2):612-623.
Inyushin MU, Arencibia-Albite F, de la Cruz, A; Vázquez-Torres R, Colon K, Sanabria P, Jiménez-Rivera CA. (2013) New Method to Visualize Neurons with DAT in Slices of Rat VTA Using Fluorescent Substrate for DAT, ASP+. Journal of Neuroscience and Neuroengineering 2, 2, pp. 98-103-106.
Arencibia-Albite F, Vázquez R, Velásquez-Martínez MC, Jiménez-Rivera CA. (2012) Cocaine sensitization inhibits the hyperpolarization-activated cation current Ih and reduces cell size in dopamine neurons of the ventral tegmental area. J Neurophysiol.107(8):2271-82.
Santos-Vera B, Vázquez-Torres R, Marrero HG, Acevedo JM, Arencibia-Albite F, Vélez-Hernández ME, Miranda JD, Jiménez-Rivera CA. (2012) Cocaine Sensitization Increases I (h)Current Channel Subunit 2 (HCN (2)) Protein Expression in Structures of the Mesocorticolimbic System. J Mol Neurosci.
Jiménez-Rivera CA, Figueroa J, Vázquez-Torres R, Vélez-Hernandez ME, Schwarz D, Velásquez-Martinez MC, Arencibia-Albite F. (2012) Presynaptic inhibition of glutamate transmission by α2 receptors in the VTA. Eur J Neurosci. 2012 35(9):1406-15.
Inyushin MU, Arencibia-Albite F, Vázquez-Torres R, Vélez-Hernández ME, Jiménez-Rivera CA. (2010) Alpha-2 noradrenergic receptor activation inhibits the hyperpolarization-activated cation current (Ih) in neurons of the ventral tegmental area. Neuroscience. 167(2):287-97.
Arencibia-Albite F, Paladini C, Williams JT, Jiménez-Rivera CA. (2007) Noradrenergic modulation of the hyperpolarization-activated cation current (Ih) in dopamine neurons of the ventral tegmental area. Neuroscience. 149(2):303-14.
Arencibia-Albite, F., and Jiménez-Rivera, CA (2003) Review of the passive properties of the neurons: Role on electrotonic changes in membrane potential. Puerto Rico Journal of Clinical Neuroscience. Vol. 3:37-46.